Cell

Expression cloning of TMEM16A as a calcium-activated chloride channel subunit.
Schroeder, B.C., Cheng, T., Jan, Y.N. and Jan, L.Y.
Cell 134 (6): 1019-1029. 19 September 2008

KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness.
Kubisch, C., Schroeder, B.C., Friedrich, T., Luetjohann, B., El-Amraoui, A., Marlin, S., Petit, C. and Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553
Cell 96 (3): 437-446. 5 February 1999

Epilepsia

Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness.
Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553, Schroeder, B.C., Kubisch, C., Friedrich, T. and Stein, V.
Epilepsia 41 (8): 1068-1069. August 2000

Human Molecular Genetics

Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.
Wollnik, B., Schroeder, B.C., Kubisch, C., Esperer, H.D., Wieacker, P. and Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553
Human Molecular Genetics 6 (11): 1943-1949. October 1997

Journal of Biological Chemistry

KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents.
Schroeder, B.C., Hechenberger, M., Weinreich, F., Kubisch, C. and Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553
Journal of Biological Chemistry 275 (31): 24089-24095. 4 August 2000

Journal of Clinical Investigation

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.
Heinze, C., Seniuk, A., Sokolov, M.V. ORCID: https://orcid.org/0000-0001-6476-9220, Huebner, A.K., Klementowicz, A.E., Szijártó, I.A. ORCID: https://orcid.org/0000-0002-0796-9450, Schleifenbaum, J. ORCID: https://orcid.org/0000-0002-3195-1517, Vitzthum, H., Gollasch, M. ORCID: https://orcid.org/0000-0003-2797-1934, Ehmke, H., Schroeder, B.C. and Hübner, C.A.
Journal of Clinical Investigation 124 (2): 675-686. 3 February 2014

Journal of the Association for Research in Otolaryngology

Early hearing loss upon disruption of Slc4a10 in C57BL/6 mice.
Huebner, A.K., Maier, H., Maul, A., Nietzsche, S., Herrmann, T., Praetorius, J. and Hübner, C.A.
Journal of the Association for Research in Otolaryngology 20 (3): 233-245. June 2019

Nature

A constitutively open potassium channel formed by KCNQ1 and KCNE3.
Schroeder, B.C., Waldegger, S., Fehr, S., Bleich, M., Warth, R., Greger, R. and Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553
Nature 403 (6766): 196-199. 13 January 2000

Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy.
Schroeder, B.C., Kubisch, C., Stein, V. and Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553
Nature 396 (6712): 687-690. 17 December 1998

Neuron

Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1.
Estevez, R., Schroeder, B.C., Accardi, A., Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553 and Pusch, M.
Neuron 38 (1): 47-59. 10 April 2003

Pfluegers Archiv European Journal of Physiology

Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.
Huebner, C.A., Schroeder, B.C. and Ehmke, H.
Pfluegers Archiv European Journal of Physiology 467 (3): 605-614. 16 January 2015

Science

A potassium channel mutation in neonatal human epilepsy.
Biervert, C., Schroeder, B.C., Kubisch, C., Berkovic, S.F., Propping, P., Jentsch, T.J. ORCID: https://orcid.org/0000-0002-3509-2553 and Steinlein, O.K.
Science 279 (5349): 403-406. 16 January 1998