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The Cl(-)-channel TMEM16A controls the generation of cochlear Ca(2+) waves and promotes the refinement of auditory brainstem networks.
Maul, A. and Jovanovic, S. and Huebner, A.K. and Strenzke, N. and Moser, T. and Rübsamen, R. and Hübner, C.A.
bioRxiv : 2021.07.05.451099. 5 July 2021


Early hearing loss upon disruption of Slc4a10 in C57BL/6 mice.
Huebner, A.K. and Maier, H. and Maul, A. and Nietzsche, S. and Herrmann, T. and Praetorius, J. and Hübner, C.A.
Journal of the Association for Research in Otolaryngology 20 (3): 233-245. June 2019


Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.
Huebner, C.A. and Schroeder, B.C. and Ehmke, H.
Pfluegers Archiv European Journal of Physiology 467 (3): 605-614. 16 January 2015


Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.
Heinze, C. and Seniuk, A. and Sokolov, M.V. and Huebner, A.K. and Klementowicz, A.E. and Szijártó, I.A. and Schleifenbaum, J. and Vitzthum, H. and Gollasch, M. and Ehmke, H. and Schroeder, B.C. and Hübner, C.A.
Journal of Clinical Investigation 124 (2): 675-686. 3 February 2014


Expression cloning of TMEM16A as a calcium-activated chloride channel subunit.
Schroeder, B.C. and Cheng, T. and Jan, Y.N. and Jan, L.Y.
Cell 134 (6): 1019-1029. 19 September 2008


Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1.
Estevez, R. and Schroeder, B.C. and Accardi, A. and Jentsch, T.J. and Pusch, M.
Neuron 38 (1): 47-59. 10 April 2003


KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents.
Schroeder, B.C. and Hechenberger, M. and Weinreich, F. and Kubisch, C. and Jentsch, T.J.
Journal of Biological Chemistry 275 (31): 24089-24095. 4 August 2000

Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness.
Jentsch, T.J. and Schroeder, B.C. and Kubisch, C. and Friedrich, T. and Stein, V.
Epilepsia 41 (8): 1068-1069. August 2000

A constitutively open potassium channel formed by KCNQ1 and KCNE3.
Schroeder, B.C. and Waldegger, S. and Fehr, S. and Bleich, M. and Warth, R. and Greger, R. and Jentsch, T.J.
Nature 403 (6766): 196-199. 13 January 2000


KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness.
Kubisch, C. and Schroeder, B.C. and Friedrich, T. and Luetjohann, B. and El-Amraoui, A. and Marlin, S. and Petit, C. and Jentsch, T.J.
Cell 96 (3): 437-446. 5 February 1999


Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy.
Schroeder, B.C. and Kubisch, C. and Stein, V. and Jentsch, T.J.
Nature 396 (6712): 687-690. 17 December 1998

A potassium channel mutation in neonatal human epilepsy.
Biervert, C. and Schroeder, B.C. and Kubisch, C. and Berkovic, S.F. and Propping, P. and Jentsch, T.J. and Steinlein, O.K.
Science 279 (5349): 403-406. 16 January 1998


Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.
Wollnik, B. and Schroeder, B.C. and Kubisch, C. and Esperer, H.D. and Wieacker, P. and Jentsch, T.J.
Human Molecular Genetics 6 (11): 1943-1949. October 1997

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