2019
Early hearing loss upon disruption of Slc4a10 in C57BL/6 mice.
Huebner, A.K., Maier, H., Maul, A., Nietzsche, S., Herrmann, T., Praetorius, J. and Hübner, C.A.
Journal of the Association for Research in Otolaryngology 20
(3): 233-245.
June 2019
2015
Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.
Huebner, C.A., Schroeder, B.C. and Ehmke, H.
Pfluegers Archiv European Journal of Physiology 467
(3): 605-614.
16 January 2015
2014
Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.
Heinze, C., Seniuk, A., Sokolov, M.V., Huebner, A.K., Klementowicz, A.E., Szijártó, I.A., Schleifenbaum, J., Vitzthum, H., Gollasch, M., Ehmke, H., Schroeder, B.C. and Hübner, C.A.
Journal of Clinical Investigation 124
(2): 675-686.
3 February 2014
2008
Expression cloning of TMEM16A as a calcium-activated chloride channel subunit.
Schroeder, B.C., Cheng, T., Jan, Y.N. and Jan, L.Y.
Cell 134
(6): 1019-1029.
19 September 2008
2003
Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1.
Estevez, R., Schroeder, B.C., Accardi, A., Jentsch, T.J. and Pusch, M.
Neuron 38
(1): 47-59.
10 April 2003
2000
KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents.
Schroeder, B.C., Hechenberger, M., Weinreich, F., Kubisch, C. and Jentsch, T.J.
Journal of Biological Chemistry 275
(31): 24089-24095.
4 August 2000
Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness.
Jentsch, T.J., Schroeder, B.C., Kubisch, C., Friedrich, T. and Stein, V.
Epilepsia 41
(8): 1068-1069.
August 2000
A constitutively open potassium channel formed by KCNQ1 and KCNE3.
Schroeder, B.C., Waldegger, S., Fehr, S., Bleich, M., Warth, R., Greger, R. and Jentsch, T.J.
Nature 403
(6766): 196-199.
13 January 2000
1999
KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness.
Kubisch, C., Schroeder, B.C., Friedrich, T., Luetjohann, B., El-Amraoui, A., Marlin, S., Petit, C. and Jentsch, T.J.
Cell 96
(3): 437-446.
5 February 1999
1998
Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy.
Schroeder, B.C., Kubisch, C., Stein, V. and Jentsch, T.J.
Nature 396
(6712): 687-690.
17 December 1998
A potassium channel mutation in neonatal human epilepsy.
Biervert, C., Schroeder, B.C., Kubisch, C., Berkovic, S.F., Propping, P., Jentsch, T.J. and Steinlein, O.K.
Science 279
(5349): 403-406.
16 January 1998
1997
Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.
Wollnik, B., Schroeder, B.C., Kubisch, C., Esperer, H.D., Wieacker, P. and Jentsch, T.J.
Human Molecular Genetics 6
(11): 1943-1949.
October 1997
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