Helmholtz Gemeinschaft

Search
Browse
Statistics
Feeds

Browse by Research Team

Export as [feed] Atom [feed] RSS 1.0 [feed] RSS 2.0
Group by: Date | Item Type | Source
Jump to: 2021 | 2019 | 2015 | 2014 | 2008 | 2003 | 2000 | 1999 | 1998 | 1997

2021

The Cl(-)-channel TMEM16A controls the generation of cochlear Ca(2+) waves and promotes the refinement of auditory brainstem networks.
Maul, A. and Jovanovic, S. and Huebner, A.K. and Strenzke, N. and Moser, T. and Rübsamen, R. and Hübner, C.A.
bioRxiv : 2021.07.05.451099. 5 July 2021

2019

Early hearing loss upon disruption of Slc4a10 in C57BL/6 mice.
Huebner, A.K. and Maier, H. and Maul, A. and Nietzsche, S. and Herrmann, T. and Praetorius, J. and Hübner, C.A.
Journal of the Association for Research in Otolaryngology 20 (3): 233-245. June 2019

2015

Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.
Huebner, C.A. and Schroeder, B.C. and Ehmke, H.
Pfluegers Archiv European Journal of Physiology 467 (3): 605-614. 16 January 2015

2014

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.
Heinze, C. and Seniuk, A. and Sokolov, M.V. and Huebner, A.K. and Klementowicz, A.E. and Szijártó, I.A. and Schleifenbaum, J. and Vitzthum, H. and Gollasch, M. and Ehmke, H. and Schroeder, B.C. and Hübner, C.A.
Journal of Clinical Investigation 124 (2): 675-686. 3 February 2014

2008

Expression cloning of TMEM16A as a calcium-activated chloride channel subunit.
Schroeder, B.C. and Cheng, T. and Jan, Y.N. and Jan, L.Y.
Cell 134 (6): 1019-1029. 19 September 2008

2003

Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1.
Estevez, R. and Schroeder, B.C. and Accardi, A. and Jentsch, T.J. and Pusch, M.
Neuron 38 (1): 47-59. 10 April 2003

2000

KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents.
Schroeder, B.C. and Hechenberger, M. and Weinreich, F. and Kubisch, C. and Jentsch, T.J.
Journal of Biological Chemistry 275 (31): 24089-24095. 4 August 2000

Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness.
Jentsch, T.J. and Schroeder, B.C. and Kubisch, C. and Friedrich, T. and Stein, V.
Epilepsia 41 (8): 1068-1069. August 2000

A constitutively open potassium channel formed by KCNQ1 and KCNE3.
Schroeder, B.C. and Waldegger, S. and Fehr, S. and Bleich, M. and Warth, R. and Greger, R. and Jentsch, T.J.
Nature 403 (6766): 196-199. 13 January 2000

1999

KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness.
Kubisch, C. and Schroeder, B.C. and Friedrich, T. and Luetjohann, B. and El-Amraoui, A. and Marlin, S. and Petit, C. and Jentsch, T.J.
Cell 96 (3): 437-446. 5 February 1999

1998

Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy.
Schroeder, B.C. and Kubisch, C. and Stein, V. and Jentsch, T.J.
Nature 396 (6712): 687-690. 17 December 1998

A potassium channel mutation in neonatal human epilepsy.
Biervert, C. and Schroeder, B.C. and Kubisch, C. and Berkovic, S.F. and Propping, P. and Jentsch, T.J. and Steinlein, O.K.
Science 279 (5349): 403-406. 16 January 1998

1997

Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.
Wollnik, B. and Schroeder, B.C. and Kubisch, C. and Esperer, H.D. and Wieacker, P. and Jentsch, T.J.
Human Molecular Genetics 6 (11): 1943-1949. October 1997

This list was generated on Mon May 20 02:23:49 2024 CEST.
Open Access
MDC Library