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Covid-19, ACE2, and the kidney

Item Type:Editorial
Title:Covid-19, ACE2, and the kidney
Creators Name:Hardenberg, J.H. and Luft, F.C.
Abstract:We are confronted with the most dramatic pandemic world-wide for the past 100 years. We are armed "to-the-teeth" compared to 1918, we know the agent, the genomic sequence, the bodily entry, the proliferation rate, the damage pathogenesis, and the very nature of our enemy. We can identify its bodily presence and our resistance to it in terms of neutralizing antibody production. Nonetheless, the disease has laid lame the great nations of the current world and crippled the less fortunate countries. The primary disease features are not the kidney. However, the entry point has much to do with renal and cardiovascular disease. The kidney is a common target of corona-virus (SARS-CoV2) disease; the longer-term consequences could be as well.
Keywords:Angiotensins, Betacoronavirus, Coronavirus Infections, Genomics, Molecular Evolution, Pandemics, Peptidyl-Dipeptidase A, Surveys and Questionnaires, Vertebrates, Viral Pneumonia, Animals
Source:Acta Physiologica
Page Range:e13539
Date:September 2020
Additional Information:Copyright © 2020 Scandinavian Physiological Society. Published by John Wiley & Sons Ltd. This article is being made freely available through PubMed Central as part of the COVID-19 public health emergency response. It can be used for unrestricted research re-use and analysis in any form or by any means with acknowledgement of the original source, for the duration of the public health emergency.
Official Publication:https://doi.org/10.1111/apha.13539
External Fulltext:View full text on PubMed Central
PubMed:View item in PubMed

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